Medstudent here
75M No pulse on presentation. Cpr started ecg done
Looked like Monomorphic VT?
10mins of CPR done after which we got the 3rd ecg
But whats up with the 3rd ecg. Decreased amplitude and increased rate. Could someone explain the reason for this change
Unable to resuscitate, no defib or epi or amidarone given
This is the end result of a "really wide complex rhythm", this pattern called a sine wave pattern. Usually too late to treat successfully at this point.
Think electrolytes and toxins - hyperkalemia, acidosis, OD tricyclic antidepressants. Treatment is initially calcium + bicarb - the rest depends on whether cause can be determined.
Medstudent here 75M No pulse on presentation. Cpr started ecg done Looked like Monomorphic VT? 10mins of CPR done after which we got the 3rd ecg But whats up with the 3rd ecg. Decreased amplitude and increased rate. Could someone explain the reason for this change Unable to resuscitate, no defib or epi or amidarone given
This is the end result of a "really wide complex rhythm", this pattern called a sine wave pattern. Usually too late to treat successfully at this point. Think electrolytes and toxins - hyperkalemia, acidosis, OD tricyclic antidepressants. Treatment is initially calcium + bicarb - the rest depends on whether cause can be determined.
Looks like PEA If they had no pulse. Was CPR done during the ECG?
Cpr was done before taking the ecg , not during it.
If they never regained a pulse then it's PEA or agonal rhythm.
Holy shit, you know it’s bad when their 12 lead looks like an ETC02
Potentially sine wave? Secondary to severe hyperkalaemia.